Cesium: For Hypotonics

ByОлена Коновалова

         Cesium (Cs)

         For hypotensives
          
    Cesium is relatively low in toxicity, its biological role in the human body is not fully understood, although it is believed to play a role in maintaining human homeostasis, especially it is effective in cases of sudden drops in blood pressure (fainting, shock states, collapse).

 
     The daily requirement for the human body is not established.
 

    When ingested, cesium is quickly absorbed from the gastrointestinal tract into the bloodstream. Cesium is evenly distributed in organs and tissues, mainly entering the intestine and being reabsorbed in its descending sections. About 80% of cesium that enters the body accumulates in muscles, 8% in the skeleton, and the remaining 12% is evenly distributed across other organs and tissues (heart, liver, blood content – up to 2.8 µg/L). In the human body, 137Cs is distributed relatively evenly and does not have a significant harmful effect. Cesium is primarily excreted through the kidneys and intestines.

 

    Biological role in the human body. The role of cesium in certain physiological processes is known. The stimulating effect of this element on circulatory functions and the effectiveness of its salts in various forms of hypotension have been established. Based on its pronounced hypertensive and vasoconstrictive effects, cesium salts were first used in 1888 by a Russian physician working in I.P. Pavlov’s laboratory, S.S. Botkin, for disorders of the cardiovascular system. He found that cesium chloride causes a long-lasting increase in blood pressure, and this effect is primarily related to the enhancement of cardiovascular activity and the constriction of peripheral vessels.

 
    Cesium, by enhancing and prolonging the action of endogenous vasomotor agents, has a positive effect in cases of hypotension of various origins. Cesium salts in optimal doses contribute to the rapid restoration of catastrophically low blood pressure in various types of shock and collapse.
 
    The adrenergic and sympathomimetic effects of cesium salts on central and peripheral adrenergic reactive structures have been established, which are particularly pronounced when the tone of the sympathetic division of the central nervous system is suppressed and in cases of catecholamine deficiency. Cesium salts are characterized mainly by a β–adrenergic stimulating effect.
 
    Cesium salts affect non-specific indicators of immunobiological resistance – they cause a significant increase in complement titers, lysozyme activity, and phagocytic activity of leukocytes. There are indications of a stimulating effect of cesium salts on the functions of hematopoietic organs. In microdoses, they stimulate erythropoiesis and leukopoiesis (by 20–25%), significantly increase the resistance of erythrocytes, and increase the hemoglobin content in them.
 
    Cesium chloride participates in gas exchange, activating the activity of oxidative enzymes, and cesium salts increase the body’s resistance to hypoxia.
 

    Synergists and antagonists of cesium. The synergist of cesium is rubidium.

 

    Signs of cesium deficiency: decreased appetite; stunted growth and development.

 

    Cesium is necessary: in shock states, fainting, collapse, hypotension, ulcerative diseases, diphtheria.

 

Dietary sources of cesium: cesium accumulation is maximal in the tissues of freshwater algae and Arctic terrestrial plants, especially lichens. High levels of cesium are observed in leafy lettuce (in the roots) and in mushrooms honey agarics.

     In animals, cesium accumulates mainly in muscles and the liver. The highest accumulation coefficient is noted in reindeer and North American waterfowl.  
 

 

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